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<br />Ineffectiveness of <br />separating restaurants <br />into smoking and <br />nonsmoking sections <br /> <br />were excluded from the study. Participants provided saliva samples at <br />the beginning and end of their work shift and also submitted to an <br />interview. The work shift had to be at least four hours in length. There <br />was a clear association between within-shift cotinine concentration and <br />smoking policy. Workers in places where customers could smoke re- <br />ported prevalence of respiratory irritation symptoms more than workers <br />in smoke-free workplaces. Exposed workers were found to have con- <br />centrations of salivary cotinine that have been associated with substan- <br />tial involuntary risks for cancer and heart disease. <br />Bates MN. Fawcett J, Dickson S, Berezowski R, <br />Garrett N. Exposure of hospitality workers to <br />environmental tobacco smoke. Tobacco Control. <br />2002; II: 125-9. <br /> <br />A University of California researcher compiled measurements of second- <br />hand smoke in 100 offices, 400 restaurants, and 600 homes; he excluded <br />those measuring substances in designated smoking areas and in smoke- <br />free establishments. Measured levels of environmental tobacco smoke <br />in restaurants were 1.6 to 2.0 times higher than in office workplaces and <br />1.5 times higher than in homes with at least one smoker. Levels of ETS <br />in bars were 3.9 to 6.1 times higher than in offices and 4.4 to 4.5 higher <br />than in homes. He also analyzed six studies that examined lung cancer <br />risk in food-service workers and controlled for active smoking and <br />certain other variables. He concluded that the high concentration of ETS <br />in restaurants was the most likely explanation for the increased risk of <br />lung cancer among food-service workers. He recommended prohibiting <br />smoking in restaurants and bars to protect workers' health. <br />Siegel M. Involuntary smoking in the restaurant <br />workplace: A review of employee exposure and <br />health effects. JArvIA. 1993;270:490-493. <br /> <br />"Levels of secondhand smoke that produce endothelial dysfunction in <br />nonsmokers' coronary arteries [changes that hinder the ability of these <br />arteries to transport blood] are typical of the high exposures encountered <br />by many nonsmokers daily in hospitality venues and automobiles." <br />Repace J. Effects of passive smoking on coronary <br />circulation. JM0/1A 2002;287:316-7. <br /> <br />Testing the effectiveness of restaurant smoking restrictions, researchers <br />measured particulate cadmium, a relatively specific marker compound <br />for ETS, in three types of sites: smoke-free, smoking-restricted, and <br />smoking-unrestricted. ETS is the major source of particulates in restau- <br />rants that allow smoking. They concluded that partial smoking restric- <br />tions reduce but do not eliminate ETS exposure in restaurants. <br />Brauer M, Mannetje A. Environmental tobacco <br />smoke exposure. AJPH. 1998; 88: 1834-6. <br /> <br />Researchers conducted a comparison of two markers of environmental <br />tobacco smoke in the smoking and no-smoking sections of restaurants at <br />the same time. They found that respirable suspended particles (the <br />